The role of aflatoxin B1 in the etiology of liver cirrhosis in Eastern Ethiopia

No Thumbnail Available

Date

2023-10

Journal Title

Journal ISSN

Volume Title

Publisher

Addis Ababa University

Abstract

Background: Liver cirrhosis is deemed to be a major cause of morbidity and mortality in Eastern Ethiopia; however, few studies have been conducted to identify its possible etiologies. For instance, in a hospital-based cross-sectional study, no definite etiologies were found in more than half of liver cirrhosis patients, suggesting a yet unidentified cause of this disease in this area. Meanwhile, as indicated in several studies, Eastern Ethiopia is a region with a diet particularly high in aflatoxin (AFB1). AFB1, a very toxic compound with sufficient evidence of carcinogenicity in humans, has been implicated as an emerging cause of liver cirrhosis. Objective: This study evaluated the role of AFB1 in the etiology of liver cirrhosis among patients attending Hiwot Fana Comprehensive Specialized University Hospital (HFCSUH), Eastern Ethiopia, in 2020-2021. Methods: A consecutive sample of 127 adult patients diagnosed with liver cirrhosis from internal medicine unit of HFCSUH were enrolled as cases, and 253 adults without any evidence of liver disease were enrolled from the same unit of the hospital as controls. Demographic, dietary, and lifestyle characteristics of the participants were collected using a structured questionnaire. Clinical data were abstracted on standardized reporting forms. Blood sample were collected and tested for hepatic enzymes, hepatitis C virus antibody, hepatitis B surface antigen, and AFB1-albumin adduct. Gluthatione S transferase M1 and T1 (GSTM1 and GSTT1) genes copy number variations were also determined by TaqManTM Assay. SPSS version 26.0 was used to compute the statistical analysis. V | P a g e Results: The etiology of liver cirrhosis was known in only 23% of patients. Sorghum consumption as a staple food was significantly associated with liver cirrhosis of unknown etiology. AFB1-albumin (AF-alb) detection rate was higher (p<0.05) in cases (75%) than in controls (64%) and with a median (interquartile range [IQR]) level of 11 pg/mg (5.5-25) and 7.0 pg/mg (4.3- 20.5), respectively (p<0.05). Exposure to high levels of AF-alb (adjusted odds ratio (AOR) = 2.0; 95% CI: 1.1–3.7) were significantly (p<0.05) associated with liver cirrhosis. The frequencies of null genotypes for GSTM1 and GSTT1 were 0.39 (95% CI: 0.32, 0.46) and 0.32 (95% CI: 0.26, 0.39), respectively. The risk of liver cirrhosis was shown to be reduced (p<0.05) in GSTT1 carriers compared to those with GSTT1 null genotypes (AOR = 0.47; 95% CI: 0.25, 0.86. Furthermore, the risk of liver cirrhosis was significantly lower in those with one copy of GSTT1 (AOR = 0.48; 95% CI: 0.25, 0.91) and a two or more combined copy of GSTT1 and GSTM1 genes (AOR = 0.38; 95% CI: 0.16, 0.91). Conclusions: Sorghum consumption was identified as a potential source of exposure in the current investigation, which implicated AFB1 exposure as a possible etiology of hepatic cirrhosis in Eastern Ethiopia with a high prevalence of cryptogenic cases. GSTT1 and GSTM1 genotypes also played a role in the risk of liver cirrhosis. To decrease the disease burden in this part of Ethiopia, plausible aflatoxin control strategies should be implemented.

Description

Keywords

Liver cirrhosis, Aflatoxins, Hepatitis B Virus, glutathione S- transferase, Ethiopia

Citation

Collections