Leishmania Aehiopica Infections in Cercopithecus Aethiops: Initiation of Infection and Immune Profiles.

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2000-06

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Addis Ababa University

Abstract

In the search of vaccines and dmgs for the control of diseases the availability of animal models is vital. Cutaneous leishmaniasis, caused by L. aethiopica is an endemic disease in Ethiopia. Availability of an animal model for L. aethiopica could be useful for studying the different aspects of the disease. In this study, monkeys were infected with an isolate of L. aethiopica to develop the primate experimental model of the disease. Twelve grivet monkeys (Cercopithecus aethiops) were trapped from cutaneous leishmaniasis (CL) nonendemic areas. Experimental infections were initiated in these animals after they were screened for natural infection. Inoculation of grivet monkeys with L. aethiopica parasite resulted either in lesion or symptomless infection. One grivet monkey produced clinical lesions following inoculation with promastigotes of diffuse cutaneous leishmaniasis (DCL) strain of L. aethiopica. Another grivet monkey produced nodules following inoculation with promastigotes of localized cutaneous leishmaniasis (LCL) strain of L. aethiopica. In addition to tlus, loss of hair at the infection site was seen in two of these animals after inoculation with LCL strain of Eo' aethiopica promastigotes. In order to. assess whether grivet monkeys have similar immune responses as humans following infection with leishmaniasis, we investigated the ill vivo and ill vitro immune responses of these animals to leishmanial antigens. Delayed type hypersensitivity (DTH) response as measured by skin testing indicated no significant response in the experimental animals. Monkeys were bled and the proliferative response of their peripheral blood mononuclear cells (PBMC) as well as IFN-y and interleukin IL-IO production were tested ill vitro in response to leishmanial antigens. The finding indicated that ill vitro lymphocyte proliferative response of infected animals to live localized cutaneous leishmaniasis strain L. aethiopica promastigotes was significantly higher than controls. However, ill vitro lymphocyte proliferative response of infected animals to live DeL strain of L. aethiopica promastigotes and killed parasites was not significantly higher compared to controls. Low level of IFN-y was produced after stimulation with leishmanial antigen. There was no detectable level ofIL-lO production after ill vitro stimulation

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Biology

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